Parkin D, Edwards R; Australasian Society for HIV Medicine. Conference.
Annu Conf Australas Soc HIV Med. 1993 Oct 28-30; 5: 96 (poster no. 43).
Department of HIV Medicine, Royal North Shore Hospital, St Leonards, NSW.
We report a case of hyperamylasemia in a 39 year old AIDS patient. He presented in May 1992 with Pneumocystis carinii pneumonia and was subsequently found to be HIV antibody positive. His CD4 Count was 32. On recovery AZT 600mg a day and Bactrim were commenced. In November 1992 CMV retinitis was diagnosed and Ganciclovir was commenced. Antiretroviral therapy was changed to ddl in November 1992. During April 1993 the patient complained of increasing abdominal discomfort and nausea. From February to April 1993 a slowly progressive rise in serum amylase was noted, attributed to pancreatitis, and ddl was ceased. In May 1993 the patient complained of xerostomia and bilateral parotid swelling was noted. The diagnosis of bilateral parotitis was made. A salivary gland nuclear medicine study confirmed bilateral parotid enlargement. Fractionation of the amylase in early May revealed 85% from the salivary glands, 15% from the pancreas. With conservative management the parotitis resolved and the serum amylase returned to near normal levels. This case serves to highlight that salivary gland dysfunction should be considered as a cause of hyperamylasemia in HIV/AIDS patients.
Publication Types:
Keywords:
- Acquired Immunodeficiency Syndrome
- Amylases
- CD4 Lymphocyte Count
- Cytomegalovirus Retinitis
- HIV Infections
- HIV Seropositivity
- Humans
- Pancreas
- Pancreatitis
- Parotid Diseases
- Parotitis
- Salivary Glands
- Xerostomia
Other ID:
UI: 102208058
From Meeting Abstracts