Dana B. Hancock, Ph.D.

Genetics, Environment & Respiratory Disease Group

Dana B. Hancock, Ph.D.
Postdoctoral IRTA Fellow

Tel (919) 541-4506
Fax (919) 541-2511
hancockd@niehs.nih.gov

Curriculum Vitae(Hancock_CV_edited.pdf)  P.O. Box 12233
Mail Drop A3-05
Research Triangle Park, North Carolina 27709
Delivery Instructions
 

Dana Hancock, Ph.D., joined the Epidemiology Branch as an IRTA postdoctoral fellow in May 2008 shortly after completing her doctoral research from Duke University.  As a genetic epidemiologist, Hancock is interested in the interplay of genetic and environmental risk factors leading to common, complex diseases.  Her doctoral training focused on interactions between candidate genes and environmental factors in Parkinson’s disease. Her postdoctoral training with Stephanie London, M.D., Dr.P.H., focuses on genetic risk factors on a genome-wide scale and corresponding gene-environment interactions in respiratory diseases, including asthma and chronic obstructive pulmonary disease. In doing so, well-known environmental factors for respiratory disease, such as cigarette smoking and air pollution, are considered. Hancock is particularly interested in the use of family-based samples to explore gene-environment interactions in such complex diseases.

Studies

• Mexico Childhood Asthma Study (http://www.niehs.nih.gov/research/atniehs/labs/epi/studies/mexico/index.cfm)
  The Mexico Childhood Asthma Study is a case-parent triad study of childhood allergic asthma in Mexico City
• The Atherosclerosis Risk in Communities (ARIC) Study (http://www.chaicore.com/aric/index.html) The ARIC Study is a prospective epidemiologic study conducted in four U.S. communities. ARIC is designed to investigate the etiology and natural history of atherosclerosis, the etiology of clinical atherosclerotic diseases and variation in cardiovascular risk factors.

Selected Publications

1. Hancock DB, Martin ER, Vance JM, Scott WK (2008). Nitric oxide synthase genes and their interactions with environmental factors in Parkinson's disease. Neurogenetics, in press.
2. Hancock DB, Martin ER, Mayhew GM, Stajich JM, Jewett R, Stacy MA, Scott BL, Vance JM, Scott WK (2008). Pesticide exposure and risk of Parkinson's disease: a family-based case-control study. BioMed Central Neurology 8(6). [Abstract] (http://www.ncbi.nlm.nih.gov/pubmed/18373838?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum)
3. Hancock DB, Martin ER, Li YJ, Scott WK (2007). Methods for interaction analyses using family-based case-control data: conditional logistic regression versus generalized estimating equations. Genetic Epidemiology 31(8):883-893.
4. Hancock DB, Martin ER, Stajich JM, Jewett R, Stacy MA, Scott BL, Vance JM, Scott WK (2007). Smoking, caffeine, and nonsteroidal anti-inflammatory drugs as protective factors in families with Parkinson disease. Archives of Neurology 64(4):576-580. [Abstract] (http://www.ncbi.nlm.nih.gov/pubmed/17420321?ordinalpos=2&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum)
5. Hancock DB, Martin ER, Fujiwara K, Stacy MA, Scott BL, Stajich JM, Jewett R, Li YJ, Hauser MA, Vance JM, Scott WK (2006). NOS2A and the modulating effect of cigarette smoking in Parkinson’s disease. Annals of Neurology 60(3):366-373. [Abstract] (http://www.ncbi.nlm.nih.gov/pubmed/16823855?ordinalpos=3&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum)

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