Cancer Etiology Study Section [CE]

[CE Membership Roster] [CE Meeting Rosters]


The Cancer Etiology Study Section reviews grant applications related to the causal agents, processes, and cells involved in early events in carcinogenesis. The areas included within CE involve gene regulation, DNA damage and repair mechanisms, chemical and viral carcinogenesis. The emphasis is on linking disciplines of chemistry and pathology on the etiology of cancer.  Specific areas covered by CE:

  • Chemical- and environmental induced carcinogenesis
  • Identification of causal agents such as xenobiotics and their ability to modulate gene regulation at the transcriptional level, RNA stability and processing, in early carcinogenesis events
  • DNA adducts, DNA damage and repair mechanisms, metabolism of endogenous and exogenous compounds that modulate early events in carcinogenesis
  • Responses to stress such as free radicals, oxidative stress and reactive oxygen species as they contribute to the carcinogenesis process
  • Contribution of non HIV/AIDS viruses to carcinogenesis


The study section with the most closely related areas of similar science listed in rank order are:

Radiation Therapeutics and Biology Study Section [RTB]
Cancer Molecular Pathobiology Study Section [CAMP]
Molecular Oncogenesis Study Section [MONC]
Cancer Genetics Study Section [CG]
Macromolecular Structure and Function Study Sections [MSFA]  
Molecular Genetics A Study Section [MGA]
Molecular Genetics B Study Section [MGB]
Molecular Genetics C Study Section [MGC]



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Last updated: November 21, 2008

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