Infection and inflammation may have a crucial role in the pathogenesis of atherosclerosis. This hypothesis is supported by an increasing number of reports on the interaction between chronic infection, inflammation, and atherogenesis. Assessment of serological and inflammatory markers of infection may be useful adjuncts in identifying those patients who are at a higher risk of developing vascular events, and in whom more aggressive treatments might be warranted.
Keywords: Stroke, risk factors, infection, Helicobacter pylori, Chlamydia pneumoniae, Cytomegalovirus, odontopathogens, atherosclerosis, therapy, antibiotics