Link to this page

Your search term(s) "graves disease" returned 17 results.

Page 1 2    Display All

Case Study: Celiac Disease: An Important Comorbidity Associated With Type 1 Diabetes. Clinical Diabetes. 26(2): 85-87. Spring 2008.

This article presents the case of a 33-year-old caucasian woman with a history of type 1 diabetes, diagnosed when she was 19 years old and complicated by microalbuminuria. Since 2003, she has been treated with insulin pump therapy. She presented with a slow, steady weight loss of 20 pounds during the previous year, with no apparent cause. The authors review the diagnostic tests conducted, the patient’s laboratory results, and the eventual determination that she should undergo a small bowel biopsy, which resulted in a diagnosis of celiac disease. After undertaking a gluten-free diet, the patient experienced less abdominal bloating and a 3-pound weight gain over the next 3 months. Her glycosylated hemoglobin (A1C) decreased from 7.2 to 6.3 percent with the dietary modifications. The authors discuss the common clinical features of celiac disease; the relationships among celiac disease, type 1 diabetes, and Graves’ disease; and the screening recommendations for celiac disease and thyroid autoimmunity in patients with type 1 diabetes. 9 references.

Full Record   Printer Friendly Version

Graves' Disease. Bethesda, MD: National Endocrine and Metabolic Diseases Information Service. 2008. 8 p.

This fact sheet, from the National Endocrine and Metabolic Diseases Information Service (NEMDIS), describes Graves’ disease, the most common cause of hyperthyroidism in the United States. Hyperthyroidism is a disorder that occurs when the thyroid gland makes more thyroid hormone than the body needs. In Graves’ disease, the immune system makes antibodies called thyroid-stimulating immunoglobulin (TSI) that attach to thyroid cells. TSI mimics the action of thyroid-stimulating hormone (TSH) and stimulates the thyroid to make too much thyroid hormone. The diagnosis and treatment of Graves’ disease is often performed by an endocrinologist, a doctor who specializes in the body’s hormone-secreting glands. The fact sheet reviews the symptoms of Graves’ disease, the condition called Graves’ ophthalmopathy, who is likely to develop Graves’ disease, diagnostic approaches to Graves’ disease, and treatment options, including radioiodine therapy, medications, surgery, and eye care. Common symptoms of hyperthyroidism include nervousness or irritability, heat intolerance, rapid and irregular heartbeat, frequent bowel movements or diarrhea, weight loss, and goiter. Graves’ ophthalmopathy is characterized by inflammation and a buildup in tissue and fat behind the eye socket, causing the eyeballs to bulge. Graves’ disease is most often treated with radioiodine therapy, which gradually destroys the cells of the thyroid gland. The fact sheet concludes with a list of six resource organizations through which readers can get more information and a brief description of the activities of the NEMDIS. 1 figure.

Full Record   Printer Friendly Version

Hyperthyroidism. Bethesda, MD: National Endocrine and Metabolic Diseases Information Service. 2008. 8 p.

This fact sheet, from the National Endocrine and Metabolic Diseases Information Service (NEMDIS), describes hyperthyroidism, a disorder that results when the thyroid gland produces more thyroid hormone than the body needs. The fact sheet is written in a question-and-answer format and covers the anatomy and function of the thyroid, the causes of hyperthyroidism, Graves’ disease, thyroid nodules, thyroiditis, iodine ingestion, overmedicating with thyroid hormone, the symptoms of hyperthyroidism, who is at risk for developing hyperthyroidism, hyperthyroidism in the aging population, diagnostic tests to confirm the presence of thyroid disease, the thyroid-stimulating hormone (TSH) test, pregnancy and hyperthyroidism, and how hyperthyroidism is treated, including antithyroid drugs, radioiodine therapy, and thyroid surgery. Some symptoms of hyperthyroidism include nervousness or irritability, fatigue or muscle weakness, trouble sleeping, heat intolerance, hand tremors, rapid and irregular heartbeat, frequent bowel movements or diarrhea, weight loss, mood swings, and goiter, an enlarged thyroid that can cause the neck to look swollen. Treatment depends on the cause and severity of the hyperthyroidism. The aim of treatment is to bring thyroid hormone levels to a normal state, thus preventing long-term complications and relieving uncomfortable symptoms. The fact sheet concludes with a list of six resource organizations through which readers can get more information and a brief description of the activities of the NEMDIS. 1 figure. 1 table.

Full Record   Printer Friendly Version

Pregnancy and Thyroid Disease. Bethesda, MD: National Endocrine and Metabolic Diseases Information Service. 2008. 8 p.

This fact sheet, from the National Endocrine and Metabolic Diseases Information Service (NEMDIS), describes pregnancy and thyroid disease. Thyroid disease is a disorder that results when the thyroid gland produces more or less thyroid hormone than the body needs. Too much thyroid hormone is called hyperthyroidism or Graves’ disease; too little thyroid hormone is called hypothyroidism. The fact sheet is written in a question-and-answer format and covers the anatomy and function of the thyroid, how pregnancy normally affects thyroid function, the causes of hyperthyroidism in pregnancy, how hyperthyroidism can affect the mother and baby, how hyperthyroidism in pregnancy is diagnosed, how hyperthyroidism is treated during pregnancy, the causes of hypothyroidism in pregnancy, how hypothyroidism can affect the mother and baby, how hypothyroidism in pregnancy is diagnosed, how hypothyroidism is treated during pregnancy, and postpartum thyroiditis. If uncontrolled during pregnancy, hyperthyroidism can be dangerous to the mother and cause health problems such as congestive heart failure and poor weight gain in the baby. Hypothyroidism during pregnancy also threatens the mother’s health and can lead to developmental disabilities in the baby. Hypothyroidism in pregnancy is safely and easily treated with synthetic thyroid hormone. The fact sheet concludes with a list of six resource organizations through which readers can get more information and a brief description of the activities of the NEMDIS. 1 figure.

Full Record   Printer Friendly Version

Endocrine Autoimmunity. IN: Gardner, D.; Shoback, D., eds. Greenspan’s Basic and Clinical Endocrinology. 8th ed. Columbus, OH: McGraw Hill. 2007. pp 59-79.

This chapter about endocrine autoimmunity is from a textbook about endocrinology that describes the scientific principles and clinical management of patients with endocrine-related diseases and disorders. The author reviews basic immunologic concepts as they apply to clinical autoimmune endocrine diseases as sole entities and as polyglandular failure syndromes. Topics include basic immune components and mechanisms, autoimmunity is multifactorial, single gland autoimmune syndromes, and autoimmune polyglandular syndromes. The most common autoimmune endocrine diseases are autoimmune thyroid disease, including hypothyroidism (Hashimoto’s disease) and hyperthyroidism (Graves’ disease), and type 1 diabetes. The chapter includes numerous black-and-white photographs and illustrations; a list of abbreviations is provided. 7 figures. 3 tables. 12 references.

Full Record   Printer Friendly Version

Graves Disease. Chevy Chase, MD: Hormone Foundation. 2007. 1 p.

This brief fact sheet provides an overview of Graves’ disease, an immune system disease that causes the thyroid gland to enlarge and results in hyperthyroidism. Written in nontechnical language, the fact sheet answers common questions about Graves’ disease, covering topics including the anatomy and function of the thyroid gland, possible symptoms of Graves’ disease, risks associated with untreated Graves’ disease, diagnostic tests used to confirm the condition, and the role of the endocrinologist. Readers are referred to the Hormone Foundation (www.hormone.org or 1–800–HORMONE) for more information. The fact sheet is also available in Spanish. 1 figure.

Full Record   Printer Friendly Version

Hormone Foundation’s Patient Guide to the Management of Maternal Hyperthyroidism Before, During and After Pregnancy. Chevy Chase, MD: Hormone Foundation. 2007. 2 p.

This fact sheet provides a patient guide to the management of maternal hyperthyroidism before, during, and after pregnancy. The guide is based on clinical guidelines written to help physicians who are evaluating and treating various types of thyroid dysfunction in pregnancy. The authors note that pregnancy, even in women with no thyroid abnormalities, causes major changes in thyroid hormone levels. This fact sheet focuses on maternal hyperthyroidism, a condition in which the mother has too much of the thyroid hormones T3 and T4, sometimes called an overactive thyroid. Readers are reminded that hyperthyroidism can have harmful effects on pregnancy, so diagnosis and treatment are vital. Most cases of hyperthyroidism during pregnancy are caused by Graves’ disease. The fact sheet outlines typical symptoms of hyperthyroidism, notes risk factors for the condition, and summarizes the recommended treatments for hyperthyroidism, notably antithyroid drug therapy. Readers are referred to the Hormone Foundation (www.hormone.org or 1–800–HORMONE) for more information. 2 figures.

Full Record   Printer Friendly Version

Disorders of the Thyroid Gland. IN: Jameson, J.L., ed. Harrison’s Endocrinology. Columbus, OH: McGraw Hill. 2006. pp 71-112.

This chapter on disorders of the thyroid gland is from a textbook that offers a comprehensive, practical look at the field of endocrinology. The thyroid hormones, thyroxine and triiodothyronine, play a critical role in cell differentiation during development and help maintain thermogenic and metabolic homeostasis in the adult. Disorders of the thyroid gland result primarily from autoimmune processes that either stimulate the overproduction of thyroid hormones––thyrotoxicosis––or cause glandular destruction and hormone deficiency––hypothyroidism. The authors discuss anatomy and development; regulation of the thyroid axis; thyroid hormone synthesis, metabolism, and action; hypothyroidism; thyrotoxicosis, including Graves' disease; thyroiditis; sick euthyroid syndrome; amiodarone effects on thyroid function; thyroid function in pregnancy; goiter and nodular thyroid disease; benign neoplasms; and thyroid cancer. The chapter includes full-color illustrations and black-and-white photographs. 13 figures. 12 tables. 15 references.

Full Record   Printer Friendly Version

Graves’ Disease. National Women’s Health Information Center. 2006. 3 p.

This fact sheet answers common questions about Graves’ disease, a type of autoimmune disease that causes overactivity of the thyroid gland, causing hyperthyroidism. The fact sheet describes the characteristics of an autoimmune disease; the symptoms of Graves’ disease; risk factors for the condition, including heredity; diagnostic tests used to confirm the presence of hyperthyroidism; treatment options; complications of Graves’ in untreated patients; and concerns for women with Graves’ who become pregnant. Treatment strategies for Graves’ disease include medications, notably antithyroid drugs; radioactive iodine, which damages thyroid cells to reduce hormone levels; and surgery, usually consisting of thyroidectomy. Patients are cautioned that they will have to take thyroid hormone supplements after any treatment that destroys the thyroid completely. Readers are referred to three resource organizations for more information: the Graves’ Disease Foundation of America (www.ngdf.org), the American Thyroid Association (www.thyroid.org), and the Thyroid Foundation of America (www.allthyroid.org).

Full Record   Printer Friendly Version

Hyperthyroidism. Jacksonville, FL: American Association of Clinical Endocrinologists (AACE). 2006. 2p.

This fact sheet reviews hyperthyroidism, a condition that develops when the body is exposed to excessive amounts of thyroid hormone. When hyperthyroidism develops, a goiter—enlargement of the thyroid—is usually present and may be associated with other symptoms including fast heart rate, anxiousness, irritability, trembling hands, weight loss, intolerance of warm temperatures, loss of scalp hair, muscle weakness, change in bowel and menstrual patterns, and protrusion of the eyes. The fact sheet reviews the causes of hyperthyroidism, including Graves' disease, toxic multinodular goiter, toxic nodule, subacute thyroiditis, postpartum thyroiditis, silent thyroiditis, excessive iodine ingestion, and overmedication with thyroid hormone. The fact sheet also explains some of the diagnostic tests that may be used to confirm hyperthyroidism and then considers treatment options, including antithyroid drugs, radioactive iodine treatment, and surgical removal of the thyroid. The fact sheet concludes that appropriate management of hyperthyroidism requires careful evaluation and ongoing care by a physician experienced in the treatment of this complex condition. Readers are referred to www.thyroidawareness.com for more information.

Full Record   Printer Friendly Version

Page 1 2    Display All

Start a new search.