| Disease/Syndrome |
Silicosis, acute |
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| Category |
Interstitial Lung Disease |
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| Acute/Chronic |
Subacute |
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| Synonyms |
Silicoproteinosis; Silica pneumoconiosis, acute; |
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| Biomedical References |
Search PubMed |
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| Comments |
Acute silicosis is rare. It develops after a brief, heavy exposure to fine silica dust with a latency period of several months. In the past the condition was observed among sandblasters and workers who tunneled through hard rock without respiratory protection. Radiographically, acute silicosis resembles alveolar proteinosis with ground glass opacities. Patients with acute silicosis rapidly deteriorate into respiratory insufficiency. [Harber, p. 382-383] [LaDou, p. 326-7] |
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| Latency/Incubation |
"Intense exposure to fine dust of high silica content over a several month period" [LaDou, p. 337] |
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| Diagnostic |
Chest x-ray; Pulmonary function test |
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| ICD-9 Code |
502 |
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| Reference Link |
OSHA - Silica |
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| Related Information in Haz-Map |
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| Symptoms/Findings |
Symptoms/Findings associated with this disease:
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| Job Tasks |
High risk job tasks associated with this disease:
- Blast, drill, remove, or crush rock, concrete or brick
- Grind or cut tiles, stones, concrete, bricks, or terrazzo
- Load or dump dusty rock, stone, or sand
- Make products from silica powder/stone or other fibrogenic minerals
- Use abrasives containing silica or silicon carbide
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| Agents |
Hazardous agents that cause the occupational disease:
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