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William T. Johnson (Thomas)

Research Chemist

Publications

Endothelial Cell-Derived Nitric Oxide Mobilization is attenuated in Copper-Deficient Rats - (Peer Reviewed Journal) - (18-Jul-08)

Cardiac Cytochrome c Oxidase Activity and Contents of Submits 1 and 4 are Altered in Offspring by Low Prenatal Intake by Rat Dams - (Peer Reviewed Journal)

Johnson, W.T., Anderson, C.M. 2008. Cardiac Cytochrome c Oxidase Activity and Contents of Submits 1 and 4 are Altered in Offspring by Low Prenatal Intake by Rat Dams. Journal of Nutrition. 138:1269-1273

Copper Deficiency Inhibits CA2+-Induced Swelling in Rat Cardiac Mitochondria - (Peer Reviewed Journal)

Johnson, W.T., Johnson, L.K. 2008. Copper Deficiency Inhibits CA2+-Induced Swelling in rat Cardiac Mitochondria. Journal of Nutritional Biochemistry.doi.10.1016/j.jnutbio.2008.02.009

Prenatal copper deficiency in rat dams causes persistent reduction in nuclear-encoded cytochrome c oxidase subunits in cardiac mitochrondria of the first generation - (Abstract)

Johnson, W.T., Anderson, C. 2008. Prenatal copper deficiency in rat dams causes persistent reduction in nuclear-encoded cytochrome c oxidase subunits in cardiac mitochrondria of the first generation [Abstract]. Journal of Federation of American Societies for Experimental Biology. 22:443.2.

Marginal copper deficiency impairs endothelium-dependent relaxation responses across two generations - (Abstract)

Anderson, C.M., Johnson, W.T. 2008. Marginal copper deficiency impairs endothelium-dependent relaxation responses across two generations [Abstract]. Journal of Federation of American Societies for Experimental Biology. 22:695.1.

Marginal copper deficiency in pregnancy: Vascular responses in dams and progeny - (Abstract)

Anderson, C.M., Johnson, W.T. 2007. Marginal copper deficiency in pregnancy: Vascular responses in dams and progeny. [abstract] Presented at Aspen Perinatal Biology Symposium, August 25-28, 2007, Aspen, CO. Abstracts were not published.

Dietary interaction of high fat and marginal copper deficiency on cardiac contractile function - (Peer Reviewed Journal)

Relling, D.P., Esberg, L.B., Johnson, W.T., Murphy, E.J., Carlson, E.C., Lukaski, H.C., Saari, J.T., Ren, J. 2007. Dietary interaction of high fat and marginal copper deficiency on cardiac contractile function. Obesity. 15(5):1242-1257.

Prenatal Cu intake by rat dams is the principle determinant of cardiac cytochrome c oxidase activity in their offspring - (Abstract)

Johnson, W.T., Anderson, C.M. 2007. Prenatal Cu intake by rat dams is the principle determinant of cardiac cytochrome c oxidase activity in their offspring [abstract]. Journal of Federation of American Societies for Experimental Biology. 21(5):A722.

Copper deficiency attenuates endothelial nitric oxide release - (Abstract)

Schuschke, D.A., Johnson, W.T., Falcone, J.C. 2007. Copper deficiency attenuates endothelial nitric oxide release [abstract]. Journal of Federation of American Societies for Experimental Biology.21(5):A721.

Copper deficiency decreases the protein expression of Complex IV but not Complex I, II, III and V in mitochondrial respiratory chain in rat heart - (Abstract)

Zeng, H., Saari, J.T., Johnson, W.T. 2007. Copper deficiency decreases the protein expression of Complex IV but not Complex I, II, III and V in mitochondrial respiratory chain in rat heart [abstract]. Journal of Federation of American Societies for Experimental Biology. 21(5):A722.

Copper deficiency decreases the protein expression of Complex IV but not Complex I, II, III, or V in mitochondrial respiratory chain in rat heart - (Peer Reviewed Journal)

Zeng, H., Saari, J.T., Johnson, W.T. 2007. Copper deficiency decreases complex IV but not complex I, II, III, or V in the mitochondrial respiratory chain in rat heart. Journal of Nutrition. 137:14-18.

Pinto beans are a source of highly bioavailable copper - (Peer Reviewed Journal)

Saari, J.T., Reeves, P.G., Johnson, W.T., Johnson, L.K. 2006. Pinto beans are a source of highly bioavailable copper. Journal of Nutrition. 136:2999-3004.

RHOA SIGNAL TRANSDUCTION: EFFECTS OF MATERNAL COPPER DEFICIENCY ON PROGENY - (Abstract)

Anderson, C.A., Brown-Borg, H.M., Johnson, W.T. 2006. Rhoa signal transduction: Effects of maternal copper deficiency on progeny [abstract]. Presented by Cindy A. Anderson at the 4th International Congress on Developmental Origins of Health and Disease (DOHaD). University of Utrecht, The Netherlands. September 12-16, 2006.

Hearts in adult offspring of copper-deficient dams exhibit decreased cytochrome c oxidase activity, increased mitochondrial hydrogen peroxide generation and enhanced formation of intracellular residual bodies - (Peer Reviewed Journal)

Johnson, W.T., Newman Jr, S.M. 2007. Hearts in adult offspring of copper-deficient dams exhibit decreased cytochrome c oxidase activity, increased mitochondrial hydrogen peroxide generation and enhanced formation of intracellular residual bodies. Journal of Nutritional Biochemistry. 18:97-104.

CARDIAC MITOCHONDRIAL FUNCTION IS ALTERED IN THE ADULT OFFSPRING OF COPPER-DEFICIENT DAMS - (Abstract)

Johnson, W.T., Newman, Jr., S.M. 2006. Cardiac mitochondrial function is altered in the adult offspring of copper-deficient dams [abstract]. Journal of Federation of American Societies for Experimental Biology. 20(5):A1065.

HIGH-FAT DIET-INDUCED JUVENILE OBESITY LEADS TO CARDIOMYOCYTE DYSFUNCTION AND UPREGULATION OF FOXO3A TRANSCRIPTION FACTOR INDEPENDENT OF LIPOTOXICITY AND APOPTOSIS - (Peer Reviewed Journal)

Relling, D.P., Esberg, L.B., Fang, C.X., Johnson, W.T., Murphy, E.J., Carlson, E.C., Saari, J.T., Ren, J. 2006. High-fat diet-induced juvenile obesity leads to cardiomyocyte dysfunction and upregulation of Foxo3a transcription factor independent of lipotoxicity and apoptosis. Journal of Hypertension. 24:549-561.

CARDIAC CYTOCHROME-C OXIDASE DEFICIENCY OCCURS DURING LATE POSTNATAL DEVELOPMENT IN PROGENY OF COPPER-DEFICIENT RATS - (Peer Reviewed Journal)

Johnson, W.T., Brown-Borg, H.M. 2006. Cardiac cytochrome-c oxidase deficiency occurs during late postnatal development in progeny of copper-deficient rats. Experimental Biology and Medicine. 231:172-180.

COPPER - (Book/Chapter)

Reeves, P.G., Johnson, W.T. 2006. Copper. In: Driskell, J.A., Wolinsky, I., editors. Sports Nutrition: Vitamins and Trace Elements, 2nd Edition. Boca Raton, FL:CRC Taylor & Francis Group. p. 235-252.

SERUM SUPEROXIDE DISMUTASE 3 (EXTRACELLULAR SUPEROXIDE DISMUTASE)ACTIVITY IS A SENSITIVE INDICATOR OF CU STATUS IN RATS - (Peer Reviewed Journal)

Johnson, W.T., Johnson, L.K., Lukaski, H.C. 2005. Serum superoxide dismutase 3 (extracellular superoxide dismutase) activity is a sensitive indicator of Cu status in rats. Journal of Nutritional Biochemistry. 16:682-692.

SERUM SUPEROXIDE DISMUTASE (SOD) ACTIVITY IS AN INDEX OF COPPER STATUS - (Abstract)

Johnson, W.T., Lukaski, H.C. 2005. Serum superoxide dismutase (SOD) activity is an index of copper status [abstract]. The Federation of American Societies for Experimental Biology Journal. 19(5):A1485.

DIETARY COPPER DEFICIENY REDUCES IRON ABSORPTION AND DUODENAL ENTEROCYTE HEPHAESTIN PROTEIN IN MALE AND FEMALE RATS - (Peer Reviewed Journal)

Reeves, P.G., DeMars, L.C.S., Johnson, W.T., Lukaski, H.C. 2005. Dietary copper deficiency reduces iron absorption and duodenal enterocyte hephaestin protein in male and female rats. Journal of Nutrition. 135:92-98.

SUPPLEMENTAL ASCORBATE OR ALPHA-TOCOPHEROL INDUCES CELL DEATH IN CU-DEFICIENT HL-60 CELLS - (Peer Reviewed Journal)

Raymond, L.J., Johnson, W.T. 2004. Supplemental ascorbate or alpha-tocopherol induces cell death in Cu-deficient HL-60 cells. Experimental Biology and Medicine. 229:885-894.

INCREASED HEME OXYGENASE-1 EXPRESSION DURING COPPER DEFICIENCY IN RATS RESULTS FROM INCREASED MITOCHONDRIAL GENERATION OF HYDROGEN PEROXIDE - (Peer Reviewed Journal)

Johnson, W.T., Demars, L.C. 2004. Increased heme oxygenase-1 expression during copper deficiency in rats results from increased mitochondrial generation of hydrogen peroxide. Journal of Nutrition. 134:1328-1333.

COPPER DEFICIENCY INDUCES HEME OXYGENASE-1 IN RAT HEART AND LIVER - (Abstract)

Johnson, W.T., Demars, L.C. 2004. Copper deficiency induces heme oxygenase-1 in rat heart and liver [abstract]. The Federation of American Societies for Experimental Biology Journal. 18:A914-A915.

CHANGES IN RESPIRATORY COMPLEX ACTIVITIES OF HEART MITOCHONDRIA CAUSED BY COPPER DEFICIENCY DURING DEVELOPMENT IN RATS ARE RESISTANT TO COPPER SUPPLEMENTATION - (Abstract)

Johnson, W.T. 2003. Changes in respiratory complex activities of heart mitochondria caused by copper deficiency during development in rats are resistant to copper supplementation [abstract]. The Federation of American Societies for Experimental Biology Journal. 17:A378.

COPPER AND BRAIN FUNCTION - (Book/Chapter)

Johnson, W.T. 2005. Copper and brain function. In: Lieberman, H.R., Kanarek, R.B., Prasad, C., editors. Nutritional Neuroscience. Boca Raton, FL:CRC Press/Taylor & Francis Group. p. 289-305.

DIETARY COPPER BUT NOT DIETARY ZINC DECREASES PROTEIN KINASE C ALPHA EXPRESSION AND DIMETHYLHYDRAZINE-INDUCED ABERRANT CRYPT FOCI FORMATION IN THE RAT COLON - (Abstract)

Fox, E., Davis, C.D., Johnson, W.T. 2002. Dietary copper but not dietary zinc decreases protein kinase C alpha expression and dimethylhydrazine-induced aberrant crypt foci formation in the rat colon [abstract]. Proceedings of the North Dakota Academy of Science. 56:45.

Last Modified: 11/07/2008

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