Title for MeetingAbstracts
Alphabetical listing of titles
HIV-1 Infections among Men who have Sex with Men (MSM) in Ecuador: Does oral sex play a role?
HIV-1 infections in Brazil: single, duals and recombinants.
HIV-1 infections undetected by antibody testing in men with urethritis in Malawi.
HIV-1 INFECTIVITY REFLECTS A DYNAMIC BALANCE BETWEEN FUSION AND ENDOCYTOSIS IN HUMAN CD4 T LYMPHOCYTES.
HIV-1 Infects placental cells and induces increase adhesion of leukocytes.
HIV-1 inhibition of hematopoiesis in vivo.
HIV-1 inhibition pattern of succinylated heparins.
HIV-1 inhibitory activity of CD8+ cell supernatants is distinct from that of RANTES, MIP-1alpha and MIP-1beta.
HIV-1 inhibitory interactions between 2',3'-dideoxyinosine (ddI) and either zidovudine (AZT), recombinant soluble CD4 (rsCD4), or recombinant interferon-alpha-a (rIFN-alpha-A).
HIV-1 inhibits CD4+, class II-restricted cytotoxic T cells (CTL) specific for herpes simplex 1 (HSV-1) via gp120.
HIV-1 inhibits macrophage-dependent CC-chemokine production by T cells via a mechanism involving induction of TGFbeta and downregulation of B7-1 expression.
HIV-1 inhibits natural killer cell function in vitro.
HIV-1 INHIBITS NK CELL FUNCTION IN VITRO.
HIV-1 integrase expressed as a fusion protein in E.coli exhibits DNA binding and endonuclease activity.
HIV-1 integrase inhibitors: discovery, structure-activity, inhibition mechanisms, selectivity.
HIV-1 integrase protein promotes reverse transcription.
HIV-1 integration in peripheral blood monocytes and cultured macrophages.
HIV-1 Interaction with Human Mannose Receptor on Astrocytes Induces Protein Dephosphorylation and Production of Matrix Metalloproteinase 2.
HIV-1 interaction with the membrane of CD4+ cells induces the nuclear translocation of 70-kDa heat shock protein (hsp70).
HIV-1 intrasubtype diversification originates from the moment of introduction of virulent HIV-1 strains into current AIDS risk groups in the 1960s and 1970s.
HIV-1 intrasubtype diversification originates from the moment of introduction of virulent HIV-1 strains into current AIDS risk groups in the 1960s and 1970s.
HIV-1 is actively produced in the female genital tract.
HIV-1 is detectable in mucosal biopsies in patients with undetectable plasma viral loads.
HIV-1 is not rapidly cytopathic for directly infected normal human T cells in vitro.
HIV-1 ISCOMS as a potent and putative vaccine against AIDS.
HIV-1 isolates from subjects on prolonged stavudine therapy remain sensitive to stavudine.
HIV-1 isolates from Zimbabwe: biologic and molecular characterization of recent Clade C isolates from seroconversion samples.
HIV-1 isolates show a dissociation of reverse transcriptase (p66/p51) and core antigen (p24) production.
HIV-1 Isolates that Exhibit Dose-Dependent NNRTI Stimulation of Reverse Transcriptase Activity and Viral Replication.
HIV-1 isolation from whole blood: standardization and applications.